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Relevant bibliographies by topics / Type 2 diabetes; Hyperglycaemia
Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022
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Contents
- Journal articles
- Dissertations / Theses
- Books
- Book chapters
- Conference papers
- Reports
Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Type 2 diabetes; Hyperglycaemia.'
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Journal articles on the topic "Type 2 diabetes; Hyperglycaemia"
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Backeström, Anna, Konstantin Papadopoulos, Sture Eriksson, Tommy Olsson, Micael Andersson, Kaj Blennow, Henrik Zetterberg, Lars Nyberg, and Olov Rolandsson. "Acute hyperglycaemia leads to altered frontal lobe brain activity and reduced working memory in type 2 diabetes." PLOS ONE 16, no.3 (March19, 2021): e0247753. http://dx.doi.org/10.1371/journal.pone.0247753.
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How acute hyperglycaemia affects memory functions and functional brain responses in individuals with and without type 2 diabetes is unclear. Our aim was to study the association between acute hyperglycaemia and working, semantic, and episodic memory in participants with type 2 diabetes compared to a sex- and age-matched control group. We also assessed the effect of hyperglycaemia on working memory–related brain activity. A total of 36 participants with type 2 diabetes and 34 controls (mean age, 66 years) underwent hyperglycaemic clamp or placebo clamp in a blinded and randomised order. Working, episodic, and semantic memory were tested. Overall, the control group had higher working memory (mean z-score 33.15 ± 0.45) than the group with type 2 diabetes (mean z-score 31.8 ± 0.44, p = 0.042) considering both the placebo and hyperglycaemic clamps. Acute hyperglycaemia did not influence episodic, semantic, or working memory performance in either group. Twenty-two of the participants (10 cases, 12 controls, mean age 69 years) were randomly invited to undergo the same clamp procedures to challenge working memory, using 1-, 2-, and 3-back, while monitoring brain activity by blood oxygen level–dependent functional magnetic resonance imaging (fMRI). The participants with type 2 diabetes had reduced working memory during the 1- and 2-back tests. fMRI during placebo clamp revealed increased BOLD signal in the left lateral frontal cortex and the anterior cingulate cortex as a function of working memory load in both groups (3>2>1). During hyperglycaemia, controls showed a similar load-dependent fMRI response, whereas the type 2 diabetes group showed decreased BOLD response from 2- to 3-back. These results suggest that impaired glucose metabolism in the brain affects working memory, possibly by reducing activity in important frontal brain areas in persons with type 2 diabetes.
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Verkleij,ChantalJ.N., Max Nieuwdorp, VictorE.A.Gerdes, Matthias Mörgelin, JoostC.M.Meijers, and PaulineF.Marx. "The effects of hyperglycaemia on thrombin-activatable fibrinolysis inhibitor." Thrombosis and Haemostasis 102, no.09 (2009): 460–68. http://dx.doi.org/10.1160/th09-01-0016.
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SummaryEpidemiological studies have shown a strong association between type 2 diabetes mellitus and cardiovascular diseases, and hypofibrinolysis may contribute to this phenomenon. The aim of this study was to determine the effect of hyperglycaemia on thrombin-activatable fibrinolysis inhibitor (TAFI). Hyperglycaemia was mimicked in vitro by incubation of TAFI with glyceraldehyde and in vivo by hyperglycaemic clamping of healthy volunteers. The effects of long-term hyperglycaemia in vivo on TAFI were investigated by comparing TAFI from poorly regulated and tightly regulated patients with type 2 diabetes. In vitro glycated TAFI showed an altered migration pattern on SDS-PAGE due to aggregation. Glycated TAFI showed decreased activity after activation by thrombin-thrombomodulin in a glyceraldehyde-dosedependent manner and a reduced anti-fibrinolytic potential. In vivo, no differences in TAFI parameters were found after hyper-glycaemic clamping of healthy volunteers and between tightly and poorly regulated patients with type 2 diabetes. Moreover, TAFI purified from poorly regulated and tightly regulated patients with type 2 diabetes migrated similarly on SDS-PAGE, indicating little or no glycation of the protein. Despite the deleterious effects of glycation of TAFI in vitro on its function, TAFI was neither affected by hyperglycaemic clamping, nor by long-term hyperglycaemia in patients with type 2 diabetes. This is in contrast to fibrinolytic factors as plasminogen-activator inhibitor I and tissue-type plasminogen activator, which are affected. We therefore hypothesise that a normally functioning TAFI under hyperglycaemic conditions may tip the haemostatic balance towards hypofibrinolysis, which may contribute to the development of cardiovascular diseases in type 2 diabetic patients.
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Praet,StephanF.E., RalphJ.F.Manders, RuthC.R.Meex, A.G.Lieverse, CoenD.A.Stehouwer, Harm Kuipers, HansA.Keizer, and LucJ.C.vanLoon. "Glycaemic instability is an underestimated problem in Type II diabetes." Clinical Science 111, no.2 (July13, 2006): 119–26. http://dx.doi.org/10.1042/cs20060041.
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The aim of the present study was to assess the level of glycaemic control by the measurement of 24 h blood glucose profiles and standard blood analyses under identical nutritional and physical activity conditions in patients with Type II diabetes and healthy normoglycaemic controls. A total of 11 male patients with Type II diabetes and 11 healthy matched controls participated in a 24 h CGMS (continuous subcutaneous glucose-monitoring system) assessment trial under strictly standardized dietary and physical activity conditions. In addition, fasting plasma glucose, insulin and HbA1c (glycated haemoglobin) concentrations were measured, and an OGTT (oral glucose tolerance test) was performed to calculate indices of whole-body insulin sensitivity, oral glucose tolerance and/or glycaemic control. In the healthy control group, hyperglycaemia (blood glucose concentration >10 mmol/l) was hardly present (2±1% or 0.4±0.2/24 h). However, in the patients with Type II diabetes, hyperglycaemia was experienced for as much as 55±7% of the time (13±2 h over 24 h) while using the same standardized diet. Breakfast-related hyperglycaemia contributed most (46±7%; P<0.01 as determined by ANOVA) to the total amount of hyperglycaemia and postprandial glycaemic instability. In the diabetes patients, blood HbA1c content correlated well with the duration of hyperglycaemia and the postprandial glucose responses (P<0.05). In conclusion, CGMS determinations show that standard measurements of glycaemic control underestimate the amount of hyperglycaemia prevalent during real-life conditions in Type II diabetes. Given the macro- and micro-vascular damage caused by postprandial hyperglycaemia, CGMS provides an excellent tool to evaluate alternative therapeutic strategies to reduce hyperglycaemic blood glucose excursions.
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Mitrakou,A. "Pathogenesis of hyperglycaemia in type 2 diabetes." Diabetes, Obesity and Metabolism 4, no.4 (July 2002): 249–54. http://dx.doi.org/10.1046/j.1463-1326.2002.00204.x.
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Yki-Järvinen, Hannele. "Toxicity of hyperglycaemia in Type 2 diabetes." Diabetes/Metabolism Reviews 14, S1 (September 1998): S45—S50. http://dx.doi.org/10.1002/(sici)1099-0895(199809)14:1+<:aid-dmr230>3.0.co;2-7.
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Yki‐Järvinen, Hannele. "Toxicity of hyperglycaemia in Type 2 diabetes." Diabetes / Metabolism Reviews 14, S1 (September 1998): S45—S50. http://dx.doi.org/10.1002/(sici)1099-0895(199809)14:1+<:aid-dmr230>3.3.co;2-z.
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Erdmann, Erland. "Cardiovascular events in patients with type 2 diabetes." British Journal of Diabetes & Vascular Disease 2, no.1_suppl (January 2002): S4—S8. http://dx.doi.org/10.1177/1474651402002001s0201.
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Diabetes is a common risk factor for cardiovascular disease. Coronary heart disease and left ventricular dysfunction are more common in diabetic patients than in non-diabetic patients, and diabetic patients benefit less from revascularisation procedures. This increased risk can only partly be explained by the adverse effects of diabetes on established risk factors; hence, a substantial part of the excess risk must be attributable to direct effects of hyperglycaemia and diabetes. In type 2 diabetes, hyperinsulinaemia, insulin resistance and hyperglycaemia have a number of potential adverse effects, including effects on endothelial function and coagulation. Risk factor modification has been shown to reduce the occurrence of cardiovascular events in patients with diabetes; indeed, diabetic patients appear to benefit more in absolute terms than non-diabetic patients. There is thus a strong case for intensive treatment of risk factors, including insulin resistance and hyperglycaemia, in patients with type 2 diabetes.
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&NA;. "Erythromycin reduces fasting hyperglycaemia in type 2 diabetes." Inpharma Weekly &NA;, no.1282 (April 2001): 13. http://dx.doi.org/10.2165/00128413-200112820-00026.
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Mohan, Sindu, DanielR.Fenton, Andrew Solomon, and JacobF.deWolff. "Hyperglycaemia in inpatients with type 2 diabetes mellitus." British Journal of Hospital Medicine 73, Sup8 (August 2012): C124—C128. http://dx.doi.org/10.12968/hmed.2012.73.sup8.c124.
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Jerreat, Lynne. "Treatment of hyperglycaemia in patients with type 2 diabetes." Nursing Standard 24, no.1 (September9, 2009): 50–57. http://dx.doi.org/10.7748/ns2009.09.24.1.50.c7261.
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Dissertations / Theses on the topic "Type 2 diabetes; Hyperglycaemia"
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Porter-Turner, Melanie Marie. "Metabolic effects and pathophysiology of glucose toxicity in type 2 diabetes mellitus." Thesis, University of Wolverhampton, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.370005.
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Konopatskaya, Olga. "Insulin-induced nitric oxide production in human endothelial cells : influence of the diabetic environment." Thesis, University of Exeter, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391835.
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Chivese, Tawanda. "Type 2 diabetes, cardiovascular risk factors and offspring overweight and obesity 5 to 6 years after hyperglycaemia first detected in pregnancy in Cape Town, South Africa." Doctoral thesis, Faculty of Health Sciences, 2021. http://hdl.handle.net/11427/33442.
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Background The number of people with type 2 diabetes mellitus (T2DM) is increasing rapidly in Africa, straining already overstretched health systems. The association between hyperglycaemia first detected in pregnancy (HFDP), which includes both diabetes mellitus in pregnancy (DIP) and gestational diabetes mellitus (GDM), and the later development of T2DM and cardiovascular disease risk in the mothers and possibly overweight in their children is well recognised. This thesis contributes to the largely unexplored body of work on the prevalence of T2DM and CVD risk factors in African women after HFDP and the relationship between HFDP and childhood overweight and obesity. The thesis investigated: 1) the prevalence of T2DM and impaired glucose metabolism in African women of childbearing age; 2) the prevalence of T2DM and cardiovascular disease risk factors in women within 6 years after HFDP, and 3) the influence of maternal blood glucose levels during pregnancy and overweight and obesity in the offspring at the preschool age. Methods A systematic review and meta-analysis of all studies published from January 2000 to 2017 was carried out to estimate the prevalence of T2DM and impaired glucose regulation states. In the PROgression to Diabetes study (PRO2D), women diagnosed with GDM using the 2008 National Institute for Health and Care Excellence (NICE) criteria during 2010 and 2011 at a major referral hospital and their offspring were reviewed up to 6 years later. Relevant maternal and foetal/neonatal data were routinely collected during pregnancy and birth. The women were recalled for an assessment of T2DM (OGTT and HbA1C) and other cardiovascular risk factors (insulin resistance, dysglycaemia, dyslipidaemia and obesity) and their offspring for overweight/obesity. The women were reclassified into DIP and GDM using the WHO 2013 criteria for the diagnosis of HFDP. The pooled prevalence of T2DM was; 7.2% (95% CI 5.6% to 8.9%), impaired fasting glycaemia, 6.0% (95% CI 4.2% to 8.2%) IGT, 0.9% to 37.0% from 39 studies in 27 African countries, and 53 075 participants. The response rate for the PRO2D was 44.2% (final sample n=220). At follow up, almost half of the women, [48% (95% CI 41.2–54.4)], had T2DM, 83% in the DIP subtype and 31% with GDM had T2DM. The type of treatment [insulin (OR 25.8, 95% CI 3.9–171.4, p = 0.001), oral antidiabetic drugs (OR 4.1, 95% CI 1.3–12.9, p = 0.018)], fasting glucose(OR 2.7, 95% CI 1.5–4.8, p = 0.001), OGTT 2-hour glucose (OR 4.3, 95% CI 2.4–7.7, p < 0.001), during pregnancy; current anthropometry [waist circumference (OR 1.1, 95% CI 1.0–1.1, p = 0.007), hip circumference (OR 0.9, 95% CI 0.8–1.0, p = 0.001), BMI (OR 1.1, 95% CI 1.0–1.3, p = 0.001)]) were associated with T2DM. The prevalence of CVD risk factors was: insulin resistance 75% (95%CI 65.9-82.3), dyslipidaemia 74.6% (95%CI 68.3- 79.9), dysglycaemia 62.3% (95%CI 55.6-68.5), and raised blood pressure 41.4% (95%CI 35.0-48.0) and metabolic syndrome 60.9% (95%CI 54.3- 67.2). Of the 443 neonates exposed to HFDP during pregnancy, almost one-third [29.6% (95%CI 25.5 – 34.0)] were large-for-gestational-age (LGA) at birth and just over a fifth [21% (95%CI 15.4 – 27.8)] were either overweight or obese at preschool age. A strong association was found between maternal fasting glucose at HFDP diagnosis and birth weight zscore (OR 1.11, 95%CI 1 -1.22, p=0.046), maternal postprandial 2-hour glucose during the third trimester and weight z-score at birth (OR 1.23, 95%CI: 1.07 - 1.42, p = 0.005) and at preschool age (OR 1.37, 95%CI: 1.03 - 1.81, p = 0.031). Conclusion The high prevalence of T2DM and CVD risk factors in relatively young women and overweight and obesity in their offspring within 6 years of the index pregnancy demonstrates the need for context-specific interventions to prevent HFDP, to optimise screening for HFDP and to reduce cardiometabolic disease risk in the postpartum period.
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Maresch, Constanze Christin [Verfasser]. "Hyperglycaemia is associated with male infertility and activin dysregulation in Type 1 Diabetes / Constanze Christin Maresch." Gießen : Universitätsbibliothek, 2017. http://d-nb.info/1137834625/34.
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Slough, Scott. "The effect of a novel series of imidazoline compounds on glucose homeostasis in the mouse." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340266.
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Sundsten, Tea. "Protein Profiling and Type 2 Diabetes." Doctoral thesis, Uppsala University, Department of Medical Cell Biology, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8458.
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Type 2 diabetes mellitus (T2DM) is a heterogeneous disease affecting millions of people worldwide. Both genetic and environmental factors contribute to the pathogenesis. The disease is characterized by alterations in many genes and their products. Historically, genomic alterations have mainly been studied at the transcriptional level in diabetes research. However, transcriptional changes do not always lead to altered translation, which makes it important to measure changes at the protein level. Proteomic techniques offer the possibility of measuring multiple protein alterations simultaneously.
In this thesis, the proteomic technique surface enhanced laser desorption ionization time-of-flight mass spectrometry (SELDI-TOF MS) has been applied and evaluated in the context of T2DM research. Protocols for pancreatic islet and serum/plasma protein profiling and identification have been developed. In addition, the technique was used to analyze the influence of genetic background versus diabetic environment by determining serum protein profiles of individuals with normal glucose tolerance (NGT) and T2DM with or without family history of diabetes. In total thirteen serum proteins displayed different levels in serum from persons with NGT versus patients with T2DM. Among these proteins, apolipoprotein CIII, albumin and one yet unidentified protein could be classified as being changed because of different genetic backgrounds. On the other hand, ten proteins for instance transthyretin, differed as a result of the diabetic environment.
When plasma protein patterns of NGT and T2DM individuals characterized by differences in early insulin responses (EIR) were compared, nine proteins were found to be varying between the two groups. Of these proteins five were identified, namely two forms of transthyretin, hemoglobin α-chain, hemoglobin β-chain and apolipoprotein H. However no individual protein alone could explain the differences in EIR. In conclusion, SELDI-TOF MS has been successfully used in the context of T2DM research to identify proteins associated with family history of diabetes and β-bell function.
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Li, Luosheng. "Molecular genetics of type 2 diabetes /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-194-2/.
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Praet, Stephan Florent Eugenie. "Exercise therapy in Type 2 diabetes." Maarsen : Maastricht : Elsevier gezondheidszorg ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=9387.
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Forbes, Shareen. "Catecholamine sensitivity preceding Type 2 diabetes." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425685.
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Gloyn, Anna Louise. "Molecular genetics of type 2 diabetes." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343364.
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Books on the topic "Type 2 diabetes; Hyperglycaemia"
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English, Patrick. Type 2 diabetes. London: Dunitz, 2001.
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Walker,RosemaryA. Type 2 Diabetes. London: Dorling Kindersley Limited, 2006.
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Stocker,ClaireJ., ed. Type 2 Diabetes. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9882-1.
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Fried, Robert, and RichardM.Carlton. Type 2 Diabetes. Boca Raton : CRC Press, 2018.: CRC Press, 2018. http://dx.doi.org/10.1201/9780429507250.
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Stocker, Claire, ed. Type 2 Diabetes. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-448-3.
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English, Patrick. Type 2 Diabetes. London: Taylor & Francis Group Plc, 2004.
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Mills, Jackie. Type 2 diabetes cookbook. Des Moines: Meredith Books, 2005.
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Type 2 diabetes explained. St. Albans, Herts: Altman, 2007.
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Rickheim, Patti. Type 2 diabetes basics. 2nd ed. Minneapolis: International Diabetes Center, 2004.
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Feinglos,MarkN., and M.AngelynBethel, eds. Type 2 Diabetes Mellitus. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-043-4.
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Book chapters on the topic "Type 2 diabetes; Hyperglycaemia"
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Brunetti,P., and G.Perriello. "Hyperglycaemia as Cardiovascular Risk in Type 2 Diabetes." In Cardiovascular Risk in Type 2 Diabetes Mellitus, 22–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59352-9_3.
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Matthews,DavidR. "Clinical management of hyperglycaemia in type 2 diabetes." In Advanced Nutrition and Dietetics in Diabetes, 103–11. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119121725.ch13.
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Chamnan, Parinya, RonaldC.W.Ma, and JulianaC.N.Chan. "Type 2 Diabetes." In Handbook of Clinical Gender Medicine, 273–82. Basel: KARGER, 2012. http://dx.doi.org/10.1159/000336414.
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Arampatzis, Adamantios, Lida Mademli, Thomas Reilly, MikeI.Lambert, Laurent Bosquet, Jean-Paul Richalet, Thierry Busso, et al. "Type 2 Diabetes." In Encyclopedia of Exercise Medicine in Health and Disease, 883. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4567.
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Nuovo, Jim. "Type 2 Diabetes." In Chronic Disease Management, 139–201. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-49369-5_7.
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Edwards,KarenL. "Type 2 Diabetes." In Gene-Environment Interactions, 285–301. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2006. http://dx.doi.org/10.1002/0471758043.ch17.
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Moore,LisaE. "Type 2 Diabetes." In Diabetes in Pregnancy, 37–44. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65518-5_5.
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Florez,HermesJ., AlexA.Sanchez, and JenniferB.Marks. "Type 2 Diabetes." In Diabetes and the Brain, 33–53. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-850-8_2.
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Heller, Lois Jane, Celette Sugg Skinner, A.JanetTomiyama, ElissaS.Epel, PeterA.Hall, Julia Allan, Lara LaCaille, et al. "Type 2 Diabetes." In Encyclopedia of Behavioral Medicine, 2012. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_101821.
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Gill,JasonM.R. "Type 2 diabetes." In Physical Activity and Health, 160–95. 3rd ed. London: Routledge, 2021. http://dx.doi.org/10.4324/9780203095270-5.
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Conference papers on the topic "Type 2 diabetes; Hyperglycaemia"
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Savira, Maya, Rusdiana, Sry Suryani Widjaja, and M.Syahputra. "Comparison of Ureum Plasma Level between Controlled Type 2 Diabetes Mellitus and Uncontrolled Type 2 Diabetes Mellitus." In International Conference of Science, Technology, Engineering, Environmental and Ramification Researches. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0010077105250527.
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Grinkevich,D., and M.Dubina. "TYPE 2 DIABETES MELLITUS IN THE ELDERLY." In SAKHAROV READINGS 2020:ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. Minsk, ICC of Minfin, 2020. http://dx.doi.org/10.46646/sakh-2020-2-46-49.
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Maalouf,NaimM., JamesC.Williams, AndrewP.Evan, JamesE.Lingeman, and JamesA.McAteer. "Type 2 Diabetes and Uric Acid Nephrolithiasis." In RENAL STONE DISEASE 2: 2nd International Urolithiasis Research Symposium. AIP, 2008. http://dx.doi.org/10.1063/1.2998021.
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Christen, Alejandra. "Arterial evaluation in type 2 diabetes mellitus." In 2010 32nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC 2010). IEEE, 2010. http://dx.doi.org/10.1109/iembs.2010.5627474.
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Rufino,RogérioL., RogérioM.Bartholo, ThiagoP.Bartholo, Claudia Costa, Agnaldo Lopes, Marília Gomes, Cátia Souza, and Andrei Melo. "Induced Sputum And Type 2 Diabetes Mellitus." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1775.
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KEATON,JACOBM., JACKLYNN.HELLWEGE, MAGGIEC.Y.NG, NICHOLETTED.PALMER, JAMESS.PANKOW, MYRIAM FORNAGE, JAMESG.WILSON, et al. "GENOME-WIDE INTERACTION WITH SELECTED TYPE 2 DIABETES LOCI REVEALS NOVEL LOCI FOR TYPE 2 DIABETES IN AFRICAN AMERICANS." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789813207813_0024.
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Chitkara, Divya, and R.K.Sharma. "Voice based detection of type 2 diabetes mellitus." In 2016 2nd International Conference on Advances in Electrical, Electronics, Information, Communication and Bio-Informatics (AEEICB). IEEE, 2016. http://dx.doi.org/10.1109/aeeicb.2016.7538402.
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Maldonado,G., C.Paredes, R.Guerrero, and C.Ríos. "FRI0645 Nailfold capillaroscopy in diabetes mellitus type 2." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.1422.
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Strazda, Gunta, Normunds Jurka, Olaga Madaja, Agnese Ozola, and Immanuels Taivans. "Association between bronchial reactivity and diabetes type 2." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4320.
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Ning Wang and Guixia Kang. "A monitoring system for type 2 diabetes mellitus." In 2012 IEEE 14th International Conference on e-Health Networking, Applications and Services (Healthcom 2012). IEEE, 2012. http://dx.doi.org/10.1109/healthcom.2012.6380067.
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Reports on the topic "Type 2 diabetes; Hyperglycaemia"
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Zaharieva, Emanuela, Elena Vikentieva, Rosica Andreeva, Dora Popova, and Zdravko Kamenov. CD36 Surface Expression on Monocytes in Type 2 Diabetes. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, April 2018. http://dx.doi.org/10.7546/crabs.2019.04.14.
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Katz, Richard, Michelle Magee, Gail Nunlee-Bland, Pamela Johnson, Linda Witkin, Carine Nassar, Clayton Bourges, et al. Comparing Three Methods to Help Patients Manage Type 2 Diabetes. Patient-Centered Outcomes Research Institute® (PCORI), July 2019. http://dx.doi.org/10.25302/4.2019.ih.13046797.
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Lai, Pei Kuan, Cheong Lieng Teng, and Feisul Idzwan Mustapha. Health Knowledge among Type 2 Diabetes Patients in Malaysia: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0044.
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Kohler, Peter, Pearl McElfish, Christopher Long, Karen Yeary, Zoran Bursac, Marie-Rachelle Narcisse, Holly Felix, et al. Developing and Testing a Type 2 Diabetes Education Program Adapted for Marshallese Culture. Patient-Centered Outcomes Research Institute® (PCORI), May 2020. http://dx.doi.org/10.25302/04.2020.ad.131007159.
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Chris Stach, Chris Stach. In plain sight: Do bacterial toxins cause cardiovascular disease and type 2 diabetes? Experiment, September 2014. http://dx.doi.org/10.18258/3434.
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Scholle, Sarah, Suzanne Morton, Daren Anderson, David Kendrick, Juell Homco, Kristin Rodriguez, Rachelle Jean-Paul, et al. Using Patient-Reported Outcome Measures during Routine Care of Patients with Type 2 Diabetes. Patient-Centered Outcomes Research Institute® (PCORI), January 2020. http://dx.doi.org/10.25302/12.2019.me.140210788.
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Chilton, Robert. Effect of glitazones on the progression of coronary artery disease in type 2 diabetes patients. Dove Press, December 2009. http://dx.doi.org/10.2147/vhrm.2.231.342.3878.
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Yin, Bei, Yiming Bi, Jingzhu Huang, and Guanjie Fan. Efficacy and Safety of Huanglian Jiedu Decoction in Type 2 Diabetes: Current State of Evidence. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0078.
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Shin, Sonya, Katrina Nelson, Letizia Trevisi, Endel John Orav, Calvin Franz, Sidney Atwood, Cameron Curley, et al. Evaluating Home-Based Health Coaching from Community Health Representatives for Navajo Adults with Type 2 Diabetes. Patient-Centered Outcomes Research Institute® (PCORI), January 2020. http://dx.doi.org/10.25302/1.2020.ad.13046566.
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Bridges, John, Ellen Janssen, Mo Zhou, Lee Bone, Jodi Segal, Tanjala Purnell, Daniel Longo, and Albert Wu. Comparing Different Ways of Asking People with Type 2 Diabetes about Their Care Priorities and Preferences. Patient‐Centered Outcomes Research Institute (PCORI)., May 2019. http://dx.doi.org/10.25302/5.2019.me.13035946.
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You might also be interested in the extended bibliographies on the topic 'Type 2 diabetes; Hyperglycaemia' for particular source types:
Journal articles Dissertations / Theses Books
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